https://pubmed.ncbi.nlm.nih.gov/?term=alzheimer's+virus
I’m not the poster you’re contempting, I’m just some asshole Empiricist who works to consistently check before contempting.
Scientific-integrity is not in the culture I grew-up in ( dad’s medical-culture Scientism has no tolerance for it, neither did mom’s Catholicism ),
but it is required for outright intellectual-integrity.
For those who won’t check the link to see the results, here is the abstract from 1 paper:
Infectious agents and Alzheimer’s disease
Abstract
Alzheimer’s disease (AD) is the leading cause of dementia worldwide. Individuals affected by the disease gradually lose their capacity for abstract thinking, understanding, communication and memory. As populations age, declining cognitive abilities will represent an increasing global health concern. While AD was first described over a century ago, its pathogenesis remains to be fully elucidated. It is believed that cognitive decline in AD is caused by a progressive loss of neurons and synapses that lead to reduced neural plasticity. AD is a multifactorial disease affected by genetic and environmental factors. The molecular hallmarks of AD include formation of extracellular β amyloid (Aβ) aggregates, neurofibrillary tangles of hyperphosphorylated tau protein, excessive oxidative damage, an imbalance of biothiols, dysregulated methylation, and a disproportionate inflammatory response. Recent reports have shown that viruses (e.g., Herpes simplex type 1, 2, 6A/B; human cytomegalovirus, Epstein-Barr virus, hepatitis C virus, influenza virus, and severe acute respiratory syndrome coronavirus 2, SARS-CoV-2), bacteria (e.g., Treponema pallidum, Borrelia burgdorferi, Chlamydia pneumoniae, Porphyromonas gingivalis, Prevotella intermedia, Tannerella forsythia, Fusobacterium nucleatum, Aggregatibacter actinomycetemcmitans, Eikenella corrodens, Treponema denticola, and Helicobacter pylori), as well as eukaryotic unicellular parasites (e.g., Toxoplasma gondii) may factor into cognitive decline within the context of AD. Microorganisms may trigger pathological changes in the brain that resemble and/or induce accumulation of Aβ peptides and promote tau hyperphosphorylation. Further, the mere presence of infectious agents is suspected to induce both local and systemic inflammatory responses promoting cellular damage and neuronal loss. Here we review the influence of infectious agents on the development of AD to inspire new research in dementia based on these pathogens.
bold-highlighting is by me.
Then they are completely-foolish.
Trump couldn’t care less about the real long-term interest of the US of A: he’s smashing the US to smithereens for sake of his importance, throughout the next 8-ish years.
( no there won’t be any replacement of him through election: it’s too late for that, now:
the Dems had their chance, in 2024, & relied on their filter-bubble & their Hopium.
The opportunity’s completely gone, & Civil War Part2 will begin roughly in 2027-9, when even the deniers are forced into seeing that it’s too late, & it should last a few years )
The Republicans’ butchering civil-rights is anti-fragile: it’ll only escalate.
& as for any proof that Trump’s values couldn’t care less about the human-lives dependent on the old way?
USAID’s gutting was the intentional severing of thousands of medical lifelines.
Extrapolate those Trump-values globally, & then you’re seeing the actual Trump, Putin’s apprentice.
Please Asia do not rely on Hopium as the Democratic Party did…
Making-believing’s the most worthless “defence” there is.
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